Publication:
lntraocular neovascularization

dc.contributor.authorYoshida, A.es
dc.contributor.authorYoshida, S.es
dc.contributor.authorIshibashi, T.
dc.contributor.authorInomata, H.
dc.date.accessioned2011-02-22T11:10:24Z
dc.date.available2011-02-22T11:10:24Z
dc.date.issued1999
dc.description.abstractAn important character of the eye is transparency, so intraocular neovascularization, which is fragile and likely to result in hemorrhage, would cause a functional disorder of the eye and contribute to loss of vision associated with such diseases as retinopathy of prematurity, diabetic retinopathy, retinal vein occlusion, and age-related macular degeneration. Recently interest in the mechanisms of intraocular neovascularization has increased, and the mechanisms have been gradually elucidated using several in vitro and in vivo angiogenesis models. Blood vessels in the eye are composed of, and surrounded by, various types of cells that produce multiple factors. Neovascularization is regulated by complex interactions among these angiogenic factors, angiostatic factors, and adhesion molecules, and some of these angiogenesis-related molecules have also been suggested as new targets for novel therapeutic agents of intraocular neo-vascularization. This review focuses on in vivo representative angiogenesis models of the corneal pocket model and the model of oxygen-induced retinopathy, and discusses the role of some angiogenesisrelated factors and adhesion molecules in intraocular neovascularization.es
dc.formatapplication/pdfes
dc.format.extent8es
dc.identifier.issn0213-3911es
dc.identifier.urihttp://hdl.handle.net/10201/19258
dc.languageenges
dc.publisherMurcia : F. Hernándezes
dc.relation.ispartofHistology and histopathologyes
dc.rightsinfo:eu-repo/semantics/openAccesses
dc.subjectNeovascularizationes
dc.subjectAdhesion moleculees
dc.subject.otherCDU::6 - Ciencias aplicadas::61 - Medicinaes
dc.titlelntraocular neovascularizationes
dc.typeinfo:eu-repo/semantics/articlees
dspace.entity.typePublicationes
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