Publication: Electron microscopic perspectives of gil1 pathology induced by 1-naphthyl-N- methylcarbamate in the goldfish (Carassius auratus Linnaeus)
Loading...
Date
1997
Authors
Pfeiffer, C.J. ; Qiu, B. ; Cho, C.H.
item.page.secondaryauthor
item.page.director
Publisher
Murcia : F. Hernández
publication.page.editor
publication.page.department
DOI
item.page.type
info:eu-repo/semantics/article
Description
Abstract
This experiment has clarified the ultrastructural
pathology, by scanning (SEM) and transmission
electron microscopy (TEM), induced by 1-
naphthyl-N- methylcarbamate (carbaryl) in the gills of
juvenile goldfish (Carassius auratus Linneaus). Carbaryl
is a low toxicity pesticide commonly used in forestry and
agriculture and for controlling aquatic weeds and
crustacean predators of shellfish, and has been known to
cause gill damage in fish and clams. A variety of cellular
changes were observed after exposure of goldfish for
96 h to a sublethal dose of 10 mg carbaryul of water.
SEM revealed secondary lamellar fusion, distortion,
thinning, and mucus release. TEM responses included
enlargement of subepithelial lymphatic spaces and
mitochondrial disruption and distortion of the lamellar
covering epithelium. Pillar cells became detached and
chloride cells were vacuolated. Fish were able to
withstand these changes in subacute experiments due to
redundacy in gill surface area.
Citation
item.page.embargo
Ir a Estadísticas
Sin licencia Creative Commons.